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Антифосфолипидный синдром - Насонов Е.Л.

Насонов Е.Л. Антифосфолипидный синдром — М.: Литтерра, 2004. — 440 c.
ISBN 5-98216-010-5
Скачать (прямая ссылка): antisindrom2004.pdf
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Статины
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2. Vaughan CJ, Gotto AM, Basson CT. The evolvingrole of statins in the management of athroscle-rosis, J Amer Col Cardiol 2000; 35:1—10.
3. Шевченко ОП, Шевченко АО. Статины. Ингибиторы ГМГ-КоА редуктазы. Москва: Реафарм, 2002; 112 с.
4. Chong PH, Seeger JD, Franklin C. Clinically relevant differences the statins: implication for therapeutic selection. Am J Med 2001; 111:390—400.
5. La Roa JC, He J, Vupputuri S. Effect of statins on risk of coronary disease. A Meta-analy-sis of randomized controlled trials. JAMA 1999; 282: 2340—2346.
6. HMG-Coa reductase inhibitors. Ed. Schmitz G, Torzewski M. Birkhauser Verlag. Basel, Boston, Berkin 2002; 151 p.
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11. Munford RS. Statins and the acute-phase response. New Engl J Med 2001; 344: 2016— 2018.
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ГЛАВА 14. Новые подходы к профилактике и лечению антифосфолипидного синдрома
13. Palinski W, Tsimikas S. Immunomodula-tory effects of statins: mechanism and potential impact on atherosclerosis. J Amer Soc Nephrol 2002; 13: 1673—1681.
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19. Kureishi Y, Luo Z, Shiojima I, et al. HMG-CoA reductase inhibitor simvastatin activates the protein kinase Akt and promotes angiogenesis m normocholesterolemic animals. Nat Med 2000; 6:1004—1010.
20. Laufs U, Marra D, Node K, et al. 3-Hydro-xy-3-methylglutaryl-CoA reductase inhibitors attenuate vascular smooth muscle proliferation by preventing rho CiTPase-induced down-regulation of p27(Kipl). J Biol Chem I999; 274: 2I926—2I93I.
21. Yang Z, Kozai T, van der Loo B, et al. HMG-CoA reductase inhibition improves endol-helial cell function and inhibits smooth muscle cell proliferation in human saphenous veins. J Am Call Cardiol. 2000; 36:1691—1697.
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